Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias. | |
Zheng Chao; Lin Xisheng; Xu Xiaolong; Wang Cheng; Zhou Jinru; Gao Bo; Fan Jing; Lu Weiguang; Hu Yaqian; Jie Qiang | |
刊名 | EBioMedicine |
2019 | |
卷号 | 40页码:695-709 |
关键词 | Achondrogenesis type 1B Atelosteogenesis type 2 SLC26A2 FGFR3 signaling Collagen secretion |
ISSN号 | 2352-3964 |
URL标识 | 查看原文 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/2826844 |
专题 | 西安交通大学 |
推荐引用方式 GB/T 7714 | Zheng Chao,Lin Xisheng,Xu Xiaolong,et al. Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias.[J]. EBioMedicine,2019,40:695-709. |
APA | Zheng Chao.,Lin Xisheng.,Xu Xiaolong.,Wang Cheng.,Zhou Jinru.,...&Yang Liu.(2019).Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias..EBioMedicine,40,695-709. |
MLA | Zheng Chao,et al."Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias.".EBioMedicine 40(2019):695-709. |
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