CORC  > 西安交通大学
Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias.
Zheng Chao; Lin Xisheng; Xu Xiaolong; Wang Cheng; Zhou Jinru; Gao Bo; Fan Jing; Lu Weiguang; Hu Yaqian; Jie Qiang
刊名EBioMedicine
2019
卷号40页码:695-709
关键词Achondrogenesis type 1B Atelosteogenesis type 2 SLC26A2 FGFR3 signaling Collagen secretion
ISSN号2352-3964
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2826844
专题西安交通大学
推荐引用方式
GB/T 7714
Zheng Chao,Lin Xisheng,Xu Xiaolong,et al. Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias.[J]. EBioMedicine,2019,40:695-709.
APA Zheng Chao.,Lin Xisheng.,Xu Xiaolong.,Wang Cheng.,Zhou Jinru.,...&Yang Liu.(2019).Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias..EBioMedicine,40,695-709.
MLA Zheng Chao,et al."Suppressing UPR-dependent overactivation of FGFR3 signaling ameliorates SLC26A2-deficient chondrodysplasias.".EBioMedicine 40(2019):695-709.
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